Verónica Rendo
- E-post:
- veronica.rendo@igp.uu.se
- Besöksadress:
- Dag Hammarskjölds väg 20
751 85 Uppsala - Postadress:
- Rudbecklaboratoriet
751 85 Uppsala

Publikationer
Senaste publikationer
Ingår i Science Translational Medicine, 2025
p21-Dependent Senescence Induction by BMP4 Renders Glioblastoma Cells Vulnerable to Senolytics
Ingår i International Journal of Molecular Sciences, 2025
- DOI för p21-Dependent Senescence Induction by BMP4 Renders Glioblastoma Cells Vulnerable to Senolytics
- Ladda ner fulltext (pdf) av p21-Dependent Senescence Induction by BMP4 Renders Glioblastoma Cells Vulnerable to Senolytics
Recurrent breakpoints in the BRD4 locus reduce toxicity associated with gene amplification
Ingår i Cell Genomics, 2025
- DOI för Recurrent breakpoints in the BRD4 locus reduce toxicity associated with gene amplification
- Ladda ner fulltext (pdf) av Recurrent breakpoints in the BRD4 locus reduce toxicity associated with gene amplification
A compendium of Amplification-Related Gain Of Sensitivity genes in human cancer
Ingår i Nature Communications, 2025
- DOI för A compendium of Amplification-Related Gain Of Sensitivity genes in human cancer
- Ladda ner fulltext (pdf) av A compendium of Amplification-Related Gain Of Sensitivity genes in human cancer
NAT2 activity increases cytotoxicity of anthracycline antibiotics and HDAC inhibitors
Ingår i Biochimica et Biophysica Acta - Molecular Basis of Disease, 2025
- DOI för NAT2 activity increases cytotoxicity of anthracycline antibiotics and HDAC inhibitors
- Ladda ner fulltext (pdf) av NAT2 activity increases cytotoxicity of anthracycline antibiotics and HDAC inhibitors
Alla publikationer
Artiklar i tidskrift
Ingår i Science Translational Medicine, 2025
p21-Dependent Senescence Induction by BMP4 Renders Glioblastoma Cells Vulnerable to Senolytics
Ingår i International Journal of Molecular Sciences, 2025
- DOI för p21-Dependent Senescence Induction by BMP4 Renders Glioblastoma Cells Vulnerable to Senolytics
- Ladda ner fulltext (pdf) av p21-Dependent Senescence Induction by BMP4 Renders Glioblastoma Cells Vulnerable to Senolytics
Recurrent breakpoints in the BRD4 locus reduce toxicity associated with gene amplification
Ingår i Cell Genomics, 2025
- DOI för Recurrent breakpoints in the BRD4 locus reduce toxicity associated with gene amplification
- Ladda ner fulltext (pdf) av Recurrent breakpoints in the BRD4 locus reduce toxicity associated with gene amplification
A compendium of Amplification-Related Gain Of Sensitivity genes in human cancer
Ingår i Nature Communications, 2025
- DOI för A compendium of Amplification-Related Gain Of Sensitivity genes in human cancer
- Ladda ner fulltext (pdf) av A compendium of Amplification-Related Gain Of Sensitivity genes in human cancer
NAT2 activity increases cytotoxicity of anthracycline antibiotics and HDAC inhibitors
Ingår i Biochimica et Biophysica Acta - Molecular Basis of Disease, 2025
- DOI för NAT2 activity increases cytotoxicity of anthracycline antibiotics and HDAC inhibitors
- Ladda ner fulltext (pdf) av NAT2 activity increases cytotoxicity of anthracycline antibiotics and HDAC inhibitors
Comparison of high-throughput single-cell RNA-seq methods for ex vivo drug screening
Ingår i NAR Genomics and Bioinformatics, 2024
- DOI för Comparison of high-throughput single-cell RNA-seq methods for ex vivo drug screening
- Ladda ner fulltext (pdf) av Comparison of high-throughput single-cell RNA-seq methods for ex vivo drug screening
Ingår i Biochemical Pharmacology, 2022
- DOI för Enhanced cytotoxicity of a novel family of ATPase inhibitors in colorectal cancer cells with high NAT2 activity
- Ladda ner fulltext (pdf) av Enhanced cytotoxicity of a novel family of ATPase inhibitors in colorectal cancer cells with high NAT2 activity
Ingår i Journal of Experimental & Clinical Cancer Research, 2021
- DOI för Common and mutation specific phenotypes of KRAS and BRAF mutations in colorectal cancer cells revealed by integrative -omics analysis
- Ladda ner fulltext (pdf) av Common and mutation specific phenotypes of KRAS and BRAF mutations in colorectal cancer cells revealed by integrative -omics analysis
Defining eligible patients for allele-selective chemotherapies targeting NAT2 in colorectal cancer
Ingår i Scientific Reports, 2020
- DOI för Defining eligible patients for allele-selective chemotherapies targeting NAT2 in colorectal cancer
- Ladda ner fulltext (pdf) av Defining eligible patients for allele-selective chemotherapies targeting NAT2 in colorectal cancer
Unexpected Acetylation of Endogenous Aliphatic Amines by Arylamine N-Acetyltransferase NAT2
Ingår i Angewandte Chemie International Edition, s. 14342-14346, 2020
- DOI för Unexpected Acetylation of Endogenous Aliphatic Amines by Arylamine N-Acetyltransferase NAT2
- Ladda ner fulltext (pdf) av Unexpected Acetylation of Endogenous Aliphatic Amines by Arylamine N-Acetyltransferase NAT2
Exploiting loss of heterozygosity for allele-selective colorectal cancer chemotherapy
Ingår i Nature Communications, 2020
- DOI för Exploiting loss of heterozygosity for allele-selective colorectal cancer chemotherapy
- Ladda ner fulltext (pdf) av Exploiting loss of heterozygosity for allele-selective colorectal cancer chemotherapy
Ingår i Oncotarget, s. 98646-98659, 2017
Somatic Ephrin Receptor Mutations Are Associated with Metastasis in Primary Colorectal Cancer
Ingår i Cancer Research, s. 1730-1740, 2017