Eva Grapengiesser

Somatostatin promotes glucose generation of Ca2+ oscillations in pancreatic islets both in the absence and presence of tolbutamide

Hellman, Bo; Dansk, Heléne; Grapengiesser, Eva

Ingår i Cell Calcium, s. 35-42, 2018

• DOI för Somatostatin promotes glucose generation of Ca2+ oscillations in pancreatic islets both in the absence and presence of tolbutamide

Sulfonylurea blockade of K_ATP channels unmasks a distinct type of glucose-induced Ca²⁺ decrease in pancreatic β-cells

Hellman, Bo; Dansk, Heléne; Grapengiesser, Eva

Ingår i Pancreas, s. 467-475, 2017

• DOI för Sulfonylurea blockade of K_ATP channels unmasks a distinct type of glucose-induced Ca²⁺ decrease in pancreatic β-cells

Small Mouse Islets Are Deficient in Glucagon-Producing Alpha Cells but Rich in Somatostatin-Secreting Delta Cells

Lau, Joey; Grapengiesser, Eva; Hellman, Bo

Ingår i Journal of Diabetes Research, 2016

• DOI för Small Mouse Islets Are Deficient in Glucagon-Producing Alpha Cells but Rich in Somatostatin-Secreting Delta Cells
• Ladda ner fulltext (pdf) av Small Mouse Islets Are Deficient in Glucagon-Producing Alpha Cells but Rich in Somatostatin-Secreting Delta Cells

Activation of alpha adrenergic and muscarinic receptors modifies early glucose suppression of cytoplasmic Ca2+ in pancreatic beta-cells

Hellman, Bo; Dansk, Helene; Grapengiesser, Eva

Ingår i Biochemical and Biophysical Research Communications - BBRC, s. 629-632, 2014

• DOI för Activation of alpha adrenergic and muscarinic receptors modifies early glucose suppression of cytoplasmic Ca2+ in pancreatic beta-cells

Glucose-induced inhibition of insulin secretion

Hellman, Bo; Grapengiesser, Eva

Ingår i Acta Physiologica, s. 479-488, 2014

• DOI för Glucose-induced inhibition of insulin secretion

Somatostatin promotes glucose generation of Ca2+ oscillations in pancreatic islets both in the absence and presence of tolbutamide

Hellman, Bo; Dansk, Heléne; Grapengiesser, Eva

Ingår i Cell Calcium, s. 35-42, 2018

• DOI för Somatostatin promotes glucose generation of Ca2+ oscillations in pancreatic islets both in the absence and presence of tolbutamide

Sulfonylurea blockade of K_ATP channels unmasks a distinct type of glucose-induced Ca²⁺ decrease in pancreatic β-cells

Hellman, Bo; Dansk, Heléne; Grapengiesser, Eva

Ingår i Pancreas, s. 467-475, 2017

• DOI för Sulfonylurea blockade of K_ATP channels unmasks a distinct type of glucose-induced Ca²⁺ decrease in pancreatic β-cells

Small Mouse Islets Are Deficient in Glucagon-Producing Alpha Cells but Rich in Somatostatin-Secreting Delta Cells

Lau, Joey; Grapengiesser, Eva; Hellman, Bo

Ingår i Journal of Diabetes Research, 2016

• DOI för Small Mouse Islets Are Deficient in Glucagon-Producing Alpha Cells but Rich in Somatostatin-Secreting Delta Cells
• Ladda ner fulltext (pdf) av Small Mouse Islets Are Deficient in Glucagon-Producing Alpha Cells but Rich in Somatostatin-Secreting Delta Cells

Activation of alpha adrenergic and muscarinic receptors modifies early glucose suppression of cytoplasmic Ca2+ in pancreatic beta-cells

Hellman, Bo; Dansk, Helene; Grapengiesser, Eva

Ingår i Biochemical and Biophysical Research Communications - BBRC, s. 629-632, 2014

• DOI för Activation of alpha adrenergic and muscarinic receptors modifies early glucose suppression of cytoplasmic Ca2+ in pancreatic beta-cells

The Neurotransmitter ATP Triggers Ca²⁺ Responses Promoting Coordination of Pancreatic Islet Oscillations

Gylfe, Erik; Grapengiesser, Eva; Dansk, Heléne; Hellman, Bo

Ingår i Pancreas, s. 258-263, 2012

• DOI för The Neurotransmitter ATP Triggers Ca²⁺ Responses Promoting Coordination of Pancreatic Islet Oscillations

Isolated mouse islets respond to glucose with an initial peak of glucagon release followed by pulses of insulin and somatostatin in antisynchrony with glucagon

Hellman, Bo; Salehi, Albert; Grapengiesser, Eva; Gylfe, Erik

Ingår i Biochemical and Biophysical Research Communications - BBRC, s. 1219-1223, 2012

• DOI för Isolated mouse islets respond to glucose with an initial peak of glucagon release followed by pulses of insulin and somatostatin in antisynchrony with glucagon

Absence of adenosine A(1) receptors unmasks pulses of insulin release and prolongs those of glucagon and somatostatin

Salehi, Albert; Parandeh, Fariborz; Fredholm, Bertil; Grapengiesser, Eva et al.

Ingår i Life Sciences, s. 470-476, 2009

• DOI för Absence of adenosine A(1) receptors unmasks pulses of insulin release and prolongs those of glucagon and somatostatin

Glucose generates coincident insulin and somatostatin pulses and anti-synchronous glucagon pulses from human pancreatic islets

Hellman, Bo; Salehi, Albert; Gylfe, Erik; Dansk, Heléne et al.

Ingår i Endocrinology, s. 5334-5340, 2009

• DOI för Glucose generates coincident insulin and somatostatin pulses and anti-synchronous glucagon pulses from human pancreatic islets

Effects of external ATP on Ca(2+) signalling in endothelial cells isolated from mouse islets

Hellman, Bo; Jansson, Leif; Dansk, Helene; Grapengiesser, Eva

Ingår i Endocrine, s. 33-40, 2007

• DOI för Effects of external ATP on Ca(2+) signalling in endothelial cells isolated from mouse islets

Insulinoscillationer - en kliniskt betydelsefull rytmik. Diabetesläkemedel bör öka den pulsatila komponenten av insulinfrisättningen: Insulin oscillations -- clinically important rhythm. Antidiabetics should increase the pulsatile component of insulin release

Hellman, Bo; Gylfe, Erik; Grapengiesser, Eva; Dansk, Helene et al.

Ingår i Läkartidningen, s. 2236-2239, 2007

Phospholipase A2 is important for glucose induction of rhythmic Ca2+ signals in pancreatic beta cells

Larsson-Nyrén, Gerd; Grapengiesser, Eva; Hellman, Bo

Ingår i Pancreas, s. 173-179, 2007

• DOI för Phospholipase A2 is important for glucose induction of rhythmic Ca2+ signals in pancreatic beta cells

Pulses of somatostatin release are slightly delayed compared with insulin and antisynchronous to glucagon

Salehi, Albert; Qader, Saleem S; Grapengiesser, Eva; Hellman, Bo

Ingår i Regulatory Peptides, s. 43-49, 2007

• DOI för Pulses of somatostatin release are slightly delayed compared with insulin and antisynchronous to glucagon

Glucose induces glucagon release pulses antisynchronous with insulin and sensitive to purinoceptor inhibition.

Grapengiesser, Eva; Salehi, Albert; Qader, Saleem S; Hellman, Bo

Ingår i Endocrinology, s. 3472-7, 2006

External ATP triggers Ca2+ signals suited for synchronization of pancreatic B-cells

Grapengiesser, Eva; Dansk, Heléne; Hellman, Bo

Ingår i Journal of Endocrinology, s. 69-79, 2005

Inhibition of purinoceptors amplifies glucose-stimulated insulin release with removal of its pulsatility.

Salehi, Albert; Qader, Saleem S; Grapengiesser, Eva; Hellman, Bo

Ingår i Diabetes, s. 2126-31, 2005

Pulses of external ATP aid to the synchronization of pancreatic B-cells by generating premature Ca2+ oscillations

Grapengiesser, Eva; Dansk, Heléne; Hellman, Bo

Ingår i Biochemical Pharmacology, s. 667-674, 2004

Effect of transforming growth factor-beta on calcium homeostasis inprostate carcinoma cells.

Gizatullina, Zemfira Z.; Grapengiesser, Eva; Shabalina, Irina G.; Nedergaard, Jan et al.

Ingår i Biochem Biophys Res Commun, s. 643, 2003

Glucose induces oscillations of cytoplasmic Ca2+, Sr2+ and Ba2+ in pancreatic beta-cells without participation of the thapsigargin-sensitive store

Liu, Y-J; Grapengiesser, Eva; Gylfe, Erik; Hellman, Bo

Ingår i Cell Calcium, s. 165-173, 1995

Glucose-induced inhibition of insulin secretion

Hellman, Bo; Grapengiesser, Eva

Ingår i Acta Physiologica, s. 479-488, 2014

• DOI för Glucose-induced inhibition of insulin secretion

Purinergic signalling in pancreatic islet endothelial cells

Jansson, Leif; Grapengiesser, Eva; Hellman, Bo

Ingår i Extracellular ATP and adenosine as regulators of endothelial cell function, s. 215-231, Springer, 2010

• DOI för Purinergic signalling in pancreatic islet endothelial cells