Hervé Nicoloff
Forskare vid Institutionen för medicinsk biokemi och mikrobiologi; Infektioner och Immunitet; Dan I Andersson
- E-post:
- herve.nicoloff@imbim.uu.se
- Besöksadress:
- BMC
Husargatan 3
752 37 UPPSALA - Postadress:
- Box 582
751 23 UPPSALA
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Kort presentation
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I am a reasercher in Dan Andersson's group. My research focuses on antibiotic resistance, with a specific interest in population genetics and genetic diversity.
Link to the group
Publikationer
Senaste publikationer
- High prevalence of heteroresistance in Staphylococcus aureus is caused by a multitude of mutations in core genes (2024)
- Three concurrent mechanisms generate gene copy number variation and transient antibiotic heteroresistance (2024)
- Impact of the pan-genome of Escherichia coli on the potential for de novo antibiotic resistance evolution (2022)
- Impact of the pan-genome of Escherichia coli on the potential for de novo antibiotic resistance evolution (2022)
- Low levels of tetracyclines select for a mutation that prevents the evolution of high-level resistance to tigecycline (2022)
Alla publikationer
Artiklar
- High prevalence of heteroresistance in Staphylococcus aureus is caused by a multitude of mutations in core genes (2024)
- Three concurrent mechanisms generate gene copy number variation and transient antibiotic heteroresistance (2024)
- Low levels of tetracyclines select for a mutation that prevents the evolution of high-level resistance to tigecycline (2022)
- Mechanisms and clinical relevance of bacterial heteroresistance (2019)
- The high prevalence of antibiotic heteroresistance in pathogenic bacteria is mainly caused by gene amplification (2019)
- A portable epigenetic switch for bistable gene expression in bacteria (2019)
- Appropriate Regulation of the sigma(E)-Dependent Envelope Stress Response Is Necessary To Maintain Cell Envelope Integrity and Stationary-Phase Survival in Escherichia coli (2017)
- Unstable tandem gene amplification generates heteroresistance (variation in resistance within a population) to colistin in Salmonella enterica (2016)
- Indirect resistance to several classes of antibiotics in cocultures with resistant bacteria expressing antibiotic-modifying or -degrading enzymes (2016)
- Lon protease inactivation, or translocation of the lon gene, potentiate bacterial evolution to antibiotic resistance (2013)
Dataset
- Impact of the pan-genome of Escherichia coli on the potential for de novo antibiotic resistance evolution (2022)
- Impact of the pan-genome of Escherichia coli on the potential for de novo antibiotic resistance evolution (2022)
- Clonal spread and antibiotic selection of a mutation preventing the evolution of high-level resistance (2022)
- Clonal spread and antibiotic selection of a mutation preventing the evolution of high-level resistance (2022)
- Clonal spread and antibiotic selection of a mutation preventing the evolution of high-level resistance (2022)
- Clonal spread and antibiotic selection of a mutation preventing the evolution of high-level resistance (2022)
- Clonal spread and antibiotic selection of a mutation preventing the evolution of high-level resistance (2022)
- Impact of the genetic background on plasmid-borne antibiotic resistance (2022)
- Impact of the genetic background on plasmid-borne antibiotic resistance (2022)
- Clonal spread and antibiotic selection of a mutation preventing the evolution of high-level resistance (2022)
- Impact of the genetic background on plasmid-borne antibiotic resistance (2022)
- Impact of the genetic background on plasmid-borne antibiotic resistance (2022)
